January 08, 2025

Blood Pressure Less Important After Age 90

"Our findings suggest that vascular risk factors are not risk factors for cerebrovascular pathologic changes at this age, medications have mitigated risks, or survival bias obscures associations."

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January 06, 2025

Female Vulnerability to Alzheimer’s Linked to Unique Genetic Pathways

Neuropathology-based genetic study links disrupted lipid metabolism and immune dysfunction to Alzheimer’s.

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December 30, 2024

Tppp3 Gene Boosts Optic Nerve Healing in Mammals, Study Finds

Researchers uncover the regenerative potential of Tppp3, a gene that enhances retinal ganglion cell survival and stimulates axon regeneration.

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December 27, 2024

The Retina as a Window to Alzheimer’s: Study Highlights Key Microglial Changes

Using advanced machine learning, researchers examine microglial morphology in Alzheimer’s, revealing novel insights into disease mechanisms.

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December 26, 2024

Study Links Dopamine Dysregulation to Chronic Pain

New research uncovers how the loss of JMJD3, a key histone demethylase, disrupts dopamine production and amplifies chronic inflammatory pain in mice, offering insights into potential therapeutic targets.

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November 07, 2024

AI in Neuropathology: How Language Models Are Transforming Brain Tumor Diagnosis

New research explores how integrating AI with clinical guidelines can enhance diagnostic precision for complex brain tumors.

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October 15, 2024

Novel Anti-Acetylated Tau Antibodies Offer New Insights and Potential Therapies for Alzheimer's Disease

With the development of monoclonal antibodies against acetylated tau, researchers unlock new possibilities for Alzheimer's diagnostics and treatment.

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September 26, 2024

TREM2 Deficiency May Speed up Myelin Damage

Scientists identify the crucial role of TREM2 in promoting remyelination and protecting brain health.

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October 14, 2024

VCP Gene Mutation in ALS and FTD Found to Cause Mitochondrial Hypermetabolism and Calcium Imbalance

New findings suggest that mitochondrial hypermetabolism and calcium overload caused by a VCP mutation play a critical role in ALS and frontotemporal dementia, opening potential therapeutic avenues.

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